Management dermatologist Hautarzt Raised, rough patch that is red to violet-colored: As the person continues to scratch or rub the itchy patch, a scaly, reddish to violet-colored patch appears.
Contagious skin diseases Geriatrics/Aging 4. Work ⁄ school 1.00 0.00-1.00 1.00 1.00-2.00 < 0.001
Triggers of neurodermatitis include: Experts argue that the observance of certain principles in nutrition significantly speeds up and facilitates recovery. Thus, a diet with dermatitis is the most important link in the successful treatment of pathology.
Twitter Williams H, Flohr C. How epidemiology has challenged 3 prevailing concepts about atopic dermatitis. J Allergy Clin Immunol. 2006 Jul. 118(1):209-13. [Medline].
Use the link below to share a full-text version of this article with your friends and colleagues. Learn more. 9. Chachaj A, Małyszczak K, Pyszel K. et al. Physical and psychological impairments of women with upper limb lymphedema following breast cancer treatment. Psychooncology. 2010 Mar;19(3):299–305. [PubMed]
J Allergy Clin Immunol. 2001; 108: 607-614 Seborrheic Dermatitis in Children Artikel Fakten
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Asthma Action Plan Heat exposure Diseases of the ear, throat and nose (otolaryngology) Clinical guidelines Also known as neurodermatitis, lichen simplex chronicus is a localized pruritic eruption that results from chronic scratching and rubbing, eventuating in a scratch–itch–scratch cycle. Clinically, lesions appear erythematous or hyperpigmented, lichenified and scaly. High-potency topical steroids are often required to break the cycle. Steroid-impregnated tape, such as flurandrenolide (Cordran), applied at bedtime or after bathing and left in place for up to12 hours, also protects the lesions from being scratched. When symptoms improve, the potency of the steroid and the frequency of use can be reduced. Topical doxepin relieves pruritus and also helps break the cycle, but systemic absorption and drowsiness sometimes limit its use. If applicable, lesions can be covered with dressings, such as an Unna boot, to prevent the patient from scratching. More nodular lesions are termed prurigo nodularis, or picker’s nodules.
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Updated: Nov 15, 2017 Neurodermatitis is characterized by decreased serum levels of prostaglandin precursors, decrease of cAMP levels in leukocytes due to the defect of beta-adrenergic receptors, and also as a result of an increased activity of phosphodiesterase. It is believed that the consequence of cAMP decreased level can be an increased release of inflammatory mediators from leukocytes, including histamine, which causes decrease in the functional activity of T-lymphocytes via H2-receptor. This may explain IgE hyperproduction. The association with certain antibodies of tissue histocompatibility (HLA-A1, A9, B12, D24, DR1, DR7 and others) is found. According to the data of P. M. Alyeva (1993) DR5 antigen is a risk factor of disease development, and DR4 and DRw6 antigens are resistance factors. Most authors consider Vidal’s disease and disseminated neurodermatitis as an independent disease, but in patients with Vidal’s disease, the detection of immune phenomena, which are peculiar to atopic dermatitis, difference absence in the distribution of histocompatibility antigens in patients with the various prevalence of the process, the similarity in the disturbance of the metabolism of biogenic amines allow to consider disseminated neurodermatitis and Vidal’s disease as manifestations of one disease.
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