annular lichen planus | oral lichen planus images

Lichen planus can be diagnosed clinically in classic cases, although biopsy is often helpful to confirm the diagnosis and is required for more atypical presentations. A 4-mm punch biopsy should be adequate on the skin or in the mouth. The histology shows a characteristic “saw-tooth” pattern of epidermal hyperplasia; hyperparakeratosis with thickening of the granular cell layer; and vacuolar alteration of the basal layer of the epidermis, with an intense infiltration (mainly T cells) at the dermal-epidermal junction. A 4-mm punch biopsy of perilesional skin for direct immunofluorescence may be added to the workup when bullous lesions, pemphigus, or bullous pemphigoid is present. Tables 2 and 3 present the differential diagnosis of cutaneous and oral lichen planus.
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+ Junctional melanocytic nevus There are several variants of cutaneous LP, which may present differently. The lesions may become large, scaly and warty (hypertrophic LP), particularly on the lower legs. New spots may appear along a site of minor skin injury such as a superficial scratch (Koebner’s phenomenon). Sometimes degeneration (atrophy) of the skin may occur as lesions persist (atrophic LP) and some patients experience an absence of sweating due to degeneration of sweat glands (anhidrosis).
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Print White area on buccal mucosa where the teeth occlude Immunohistochemistry confirmed overexpression of 14–3-3 zeta and theta in premalignant oral lesions and oral squamous cell carcinoma tissues in comparison with normal epithelium [27]. Since oral lichen planus is considered a premalignant lesion, there is a possibility that this protein might be increased in this disease.
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Next article >> Oral Lichen Planus Symptoms Lichen planus, an inflammatory disease, tends to affect the skin and oral mucosa but may involve the nails, scalp, esophagus, and anogenital regions. Its various clinical presentations and treatment options are reviewed, including available data to guide management.
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Kumar, V., Abbas, A. K., & Aster, J. C. (2013). Robbins Basic Pathology. Elsevier Health Sciences. Saliva samples from the OLP and control groups were pooled using an equal amount of protein from each sample within each group. Representative gels from the OLP and healthy control groups are shown in Fig. 1. Over 100 protein spots were detected on Coomassie Blue-stained gels; image analysis identified 31 protein spots that exhibited ≥1.5-fold difference in abundance or were found exclusively in either the OLP or healthy control groups. Of these 31 protein spots, 14 protein spots (likely representing nine or 10 unique proteins) were found in both the OLP and healthy control groups (Table 2). Two spots (numbers 135 and 152) could not be identified as either a cystatin S precursor or cystatin SA. In addition, 11 protein spots (representing seven unique proteins) were detected only in the OLP group (Table 3), and six other protein spots (representing five unique proteins) were present only in the healthy control group (Table 4). In the 14 matched protein spots that were found in both the OLP and healthy control groups, seven protein spots (representing six unique proteins) exhibited increased expression in the OLP group, whereas seven other protein spots (likely representing three or four unique proteins) exhibited decreased expression in the OLP group.
+ Angioma serpiginosum 21. Sensitivity to hot, acidic or spicy foods
Lehman, JS, Tollefson, MM, Gibson, LE. ” Lichen Planus”. Int J Dermatol. vol. 48. 2009. pp. 682-94. MENU A Nature Research Journal GME Training
History[edit] X UCLA Dermatology Diabetes Nature Korea + Arteriovenous malformation Penile lichen planus
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Contact Your Representatives Aspetto bucato delle papule, associato a verruche pruriginose (arti) Share on Google Plus
Lichen planus is an itchy skin rash that is caused by an immune response. It can occur anywhere on your skin. Trending on MedicineNet
PSORIASIS An autoimmune reaction, meaning the body’s own defense system, the immune system, attacks your mouth and skin cells by mistake
Early, scattered lesions of lichen planus. 68 SNOMED-CT Switch Edition + Supranumerary nipple + Giant melanocytic nevus
Moles Lichen planus on the hands R/ 1. Bethamethason 0.1% zalf 15 g
Technology and Finance Human salivary proteins that were identified exclusively in oral lichen planus patients Lichen planus on the feet. The reticular white scale of Wickham striae is visible on some of the plaques.
Some people who suffer from oral lichen planus have lesions and sores on other parts of their bodies such as: Feedback on: Sex
1 (4.15%) Another clue to the diagnosis is that lesions may occur in areas exposed to trauma, such as lacerations. This tendency is known as an isomorphic response, or Koebner’s phenomenon. Lesions often resolve with intense hyperpigmentation. Some physicians describe lichen planus with the six “Ps”: pruritic, polygonal, planar (flat-topped), purple papules and plaques. While some patients may be asymptomatic, most experience intense pruritus, a hallmark of lichen planus.
purplish-colored lesions or bumps with flat tops on your skin or genitals FAQ Academics Methods This information provides a general overview and may not apply to everyone. Talk to your family doctor to find out if this information applies to you and to get more information on this subject.
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Mirowski GW, et al. Oral lichen planus: Pathogenesis, clinical features, and diagnosis. Accessed Feb. 5, 2016.
145, 189, 190, 205 Oral metronidazole (mechanism is unknown)
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7 Replies to “annular lichen planus | oral lichen planus images”

  1. + Acute hematopoietic malignancies
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    There is some evidence of an association between LP (especially with oral LP) and hepatitis C virus infection and blood testing may be ordered by your physician.
    16.5, 14.1
    The following factors can cause you to develop the condition. They can also be triggers in people who already have the condition:
    + Disseminated intravascular coagulopathy (DIC)
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    10% dei casi: il lichen ruber planus causa macchie bianche che si espandono ad anello

  2. 2/27/2018MACRA/MIPS Update
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    + Antinuclear antibodies (ANA)
    The rash appears abruptly and usually lasts for several months.

  3. White net-like patches or ulceration of mucous membranes, nail deformity, scarring alopecia, and other types of skin lesions may also occur.
    The differential diagnosis of cutaneous LP includes the following entities: 1) lichenoid drug eruptions (tissue or serum eosinophilia, photo-exposed areas, confluent or widespread, greater degree of parakeratosis, exposure to ACE inhibitors, antimalarials, ß-blockers, methyldopa, NSAIDs, or penicillamine); 2) Lupus erythematosus (+ANA, IgM and IgG deposits in Civatte bodies, deep nodular perivascular lymphoplasmacytic infiltrates, fat lobule necrosis); 3) lichen nitidus (small, pin-point sized, flesh colored, nonpruritic papules); 4) guttate psoriasis (prominent extensor surface involvement, no oral lesions, white silvery scale).
    + Chondroid syringoma
    Penile lichen planus

  4. + Amalgam tatoo
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    Larsen KR, Johansen JD, Reibel J, Zachariae C, Rosing K, Pedersen AML. Oral symptoms and salivary findings in oral lichen planus, oral lichenoid lesions and stomatitis. BMC Oral Health. 2017;17(1):103.View ArticlePubMedPubMed CentralGoogle Scholar
    + Allergic contact dermatitis, acute
    Extremities (face, dorsal hands, arms, and nape of neck).[a] This is more common in Middle Eastern countries in spring and summer, where sunlight appears to have a precipitating effect.[14][19][20]

  5. Contributions
    + Disorders of elastic fibres
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    Liquid chromatography coupled with tandem mass spectrometry
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    Possible Causes of a Rash on Your Wrist

  6. Histology: hair follicles are surrounded by a bandlike lymphocytic infiltrate
    For oral lichen planus, stop smoking, avoid alcohol, maintain good oral hygiene and avoid any foods that seem to irritate your mouth.
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    Although the exact etiology of lichen planus is unknown, an immune-mediated pathogenesis is recognized.1 A meta-analysis of primarily case-control studies conducted in multiple countries found a statistically significant association between hepatitis C virus (HCV) infection and lichen planus,3 although there is no known explanation for this association. Compared with the control group, patients with lichen planus had a greater prevalence of HCV exposure (odds ratio = 5.4; 95% confidence interval, 3.5 to 8.3), and patients with HCV infection had an increased prevalence of lichen planus (odds ratio = 2.5; 95% confidence interval, 2.0 to 3.1).3 It is appropriate to screen all patients with lichen planus for HCV infection.4 Patients should be asked about HCV risk factors, and liver enzyme and HCV antibody tests should be ordered.
    Le Cleach, Laurence, and Olivier Chosidow. “Lichen Planus.” The New England Journal of Medicine 366 (2012): 723-732.
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    The exclusion criteria (for both tests and controls) were: immunologic diseases, diabetes mellitus, malignancies, history of radiotherapy in the head and neck region, smoking, diseases that decrease saliva flow (such as Sjogren’s syndrome), use of anti-Parkinson’s medications, use of immunosuppressants or immunomodulators, use of medications causing xerostomia (antihistamines, anticholinergics, anticonvulsants, muscle relaxants, tranquilizers, sleeping pills, cytotoxic drugs and three-cyclic anti-depressants), patients with lesions next to amalgam fillings, lichenoid drug reactions, pregnancy or nursing, inflammatory diseases, or oral fungal diseases. Patients with evidence of dysplasia in their pathology report were also excluded.
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  7. Natural History Study Patient Registries
    We evaluated saliva from 24 OLP patients and 24 age-matched healthy control subjects. The characteristics and clinical features of these subjects are summarized in Table 6. Salivary levels of complement component C3c, fibrinogen fragment D, and cystatin SA are also described in Table 6. Salivary levels of complement component C3c exhibited significant elevation in the OLP group, compared with the healthy control group (p = 0.041) (Table 6). There was weak statistical evidence that salivary levels of fibrinogen fragment D were higher, and salivary levels of cystatin SA were lower, in the OLP group, compared with the healthy control group (p = 0.398 and p = 0.281 for the respective comparisons).

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